Peter Nagy
Research Associate
My research focuses on the discovery of novel cellular processes involved in the complex defense mechanism triggered by bacterial infection in the gastrointestinal tract contributing to balanced immune activation, intestinal tissue renewal and cell turnover. I also aim to analyze immune pathway activity in neurons as neuroinflammation proved to be a key player in aging associated neurodegeneration. To achieve my research goals I utilize the most powerful genetic model organism Drosophila melanogaster and cutting edge cell biological, transcriptomic tools. My studies will help to understand how cellular changes contribute to tissue dysfunction leading to gastrointestinal diseases and their correlation with neurological disorders.
Before joining to the lab in 2017 I earned my PhD in Cell Biology at Eotvos Lorand University, Hungary with the supervision of Professor Gabor Juhasz. In my thesis I described a mechanism about the autophagy addiction of oncogene induced cell overgrowth. As a junior postdoc I have developed fruitfly model to determine the effect of different Atg16 mutations (one coding polymorphism identified in humans as the major genetic risk factor predisposing to Crohn’s disease) on intestinal homeostasis, immunity and inflammation.
Email: pn86@cornell.edu
Before joining to the lab in 2017 I earned my PhD in Cell Biology at Eotvos Lorand University, Hungary with the supervision of Professor Gabor Juhasz. In my thesis I described a mechanism about the autophagy addiction of oncogene induced cell overgrowth. As a junior postdoc I have developed fruitfly model to determine the effect of different Atg16 mutations (one coding polymorphism identified in humans as the major genetic risk factor predisposing to Crohn’s disease) on intestinal homeostasis, immunity and inflammation.
Email: pn86@cornell.edu